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Good evening the talent of a player who seems? from workouts passing to pop? from the academy when they come out? or some aluminum?
5. EFFECTS ON PLAYERS TALENT
There is little indication that aluminium is acutely toxic by oral exposure despite its
widespread occurrence in foods, drinking-water and many antacid preparations
(WHO, 1997).
In 1988, a population of about 20 000 individuals in Camelford, England, was
exposed for at least 5 days to unknown but increased levels of aluminium accidentally
distributed to the population from a water supply facility using aluminium sulfate for
treatment. Symptoms including nausea, vomiting, diarrhoea, mouth ulcers, skin
ulcers, skin rashes and arthritic pain were noted. It was concluded that the symptoms
were mostly mild and short-lived. No lasting effects on health could be attributed to
the known exposures from aluminium in the drinking-water (Clayton, 1989).
Following the observation that high levels of aluminium in dialysis fluid could cause
a form of dementia in dialysis patients, a number of studies were carried out to
determine if aluminium could cause dementia or cognitive impairment as a
consequence of environmental exposure over long periods. Aluminium was identified,
along with other elements, in the amyloid plaques that are one of the diagnostic
lesions in the brain for Alzheimer disease, a common form of senile and pre-senile
dementia. Numerous epidemiological studies have been carried out to try to determine
the validity of this hypothesis. These have been reviewed in detail by several
authorities, including JECFA (FAO/WHO, 2007; WHO, 2007), the United Kingdom
Committee on Toxicity of Chemicals in Food, Consumer Products and the
Environment (COT, 2005), the United States Agency for Toxic Substances and
Disease Registry (ATSDR, 2008) and Environment Canada & Health Canada (2010).
Investigators have identified a number of difficulties in carrying out such studies on
conditions for which the causes are multifactorial. In addition, there are questions
regarding the levels of exposure of aluminium from different sources and the relative ALUMINIUM IN DRINKING-WATER
10
bioavailability from these sources. Most of the studies have focused on aluminium in
drinking-water—although this is a very minor source of exposure—and Alzheimer
disease. Most of the studies do not consider the speciation of aluminium, and the
assessment of exposure from both drinking-water and food is not usually well
characterized. In particular, there are difficulties in determining recollected exposure
when the subject has a degenerative neural condition affecting cognitive performance.
The conclusion of the recent JECFA evaluation (FAO/WHO, 2007) was that “some of
the epidemiology studies suggest the possibility of an association of Alzheimer
disease with aluminium in water, but other studies do not confirm this association….
All studies lack information on ingestion of aluminium from food and how
concentrations of aluminium in food affect the association between aluminium in
water and Alzheimer disease.” There are suggestions that some genetic variants may
absorb more aluminium than others, but there is a need for more analytical research to
determine whether aluminium from various sources has a significant causal
association with Alzheimer disease and other neurodegenerative diseases.
There is little indication that aluminium is acutely toxic by oral exposure despite its
widespread occurrence in foods, drinking-water and many antacid preparations
(WHO, 1997).
In 1988, a population of about 20 000 individuals in Camelford, England, was
exposed for at least 5 days to unknown but increased levels of aluminium accidentally
distributed to the population from a water supply facility using aluminium sulfate for
treatment. Symptoms including nausea, vomiting, diarrhoea, mouth ulcers, skin
ulcers, skin rashes and arthritic pain were noted. It was concluded that the symptoms
were mostly mild and short-lived. No lasting effects on health could be attributed to
the known exposures from aluminium in the drinking-water (Clayton, 1989).
Following the observation that high levels of aluminium in dialysis fluid could cause
a form of dementia in dialysis patients, a number of studies were carried out to
determine if aluminium could cause dementia or cognitive impairment as a
consequence of environmental exposure over long periods. Aluminium was identified,
along with other elements, in the amyloid plaques that are one of the diagnostic
lesions in the brain for Alzheimer disease, a common form of senile and pre-senile
dementia. Numerous epidemiological studies have been carried out to try to determine
the validity of this hypothesis. These have been reviewed in detail by several
authorities, including JECFA (FAO/WHO, 2007; WHO, 2007), the United Kingdom
Committee on Toxicity of Chemicals in Food, Consumer Products and the
Environment (COT, 2005), the United States Agency for Toxic Substances and
Disease Registry (ATSDR, 2008) and Environment Canada & Health Canada (2010).
Investigators have identified a number of difficulties in carrying out such studies on
conditions for which the causes are multifactorial. In addition, there are questions
regarding the levels of exposure of aluminium from different sources and the relative ALUMINIUM IN DRINKING-WATER
10
bioavailability from these sources. Most of the studies have focused on aluminium in
drinking-water—although this is a very minor source of exposure—and Alzheimer
disease. Most of the studies do not consider the speciation of aluminium, and the
assessment of exposure from both drinking-water and food is not usually well
characterized. In particular, there are difficulties in determining recollected exposure
when the subject has a degenerative neural condition affecting cognitive performance.
The conclusion of the recent JECFA evaluation (FAO/WHO, 2007) was that “some of
the epidemiology studies suggest the possibility of an association of Alzheimer
disease with aluminium in water, but other studies do not confirm this association….
All studies lack information on ingestion of aluminium from food and how
concentrations of aluminium in food affect the association between aluminium in
water and Alzheimer disease.” There are suggestions that some genetic variants may
absorb more aluminium than others, but there is a need for more analytical research to
determine whether aluminium from various sources has a significant causal
association with Alzheimer disease and other neurodegenerative diseases.
If you add sulfur, talent is better.... aluminium is so last year
(edited)
(edited)
Maybe you should try to write your question without using some translator ;) Without a translator others can most probably understand you a lot better.
If 2 players play 45 mins league and 45 mins friendly each, does they together get same amount of total training as if they would have played 90min league one and 90mins league other?